Hydrocarbon ingestion
- Because of their low viscosity and high volatility, hydrocarbons may be
aspirated during ingestion and cause a
secondary pneumonitis,
although most children who ingest hydrocarbons do not develop respiratory
difficulties.
- onset of respiratory symptoms and radiographic findings may be delayed.
- Patients who are asymptomatic
after 6 hours of observation and have negative radiographic findings
are unlikely to develop symptoms.
- Those who do develop clinical pneumonitis may experience dyspnea and
hypoxia and require oxygen treatment.
- Occasionally, ventilatory assistance is necessary.
- Rarely, there is a rapid progression to respiratory failure and
death.
- Acute complications of
pneumonitis
include pneumothorax, subcutaneous emphysema of the chest wall, and
pleural effusions.
- A secondary pneumonia may
develop during the ensuing week.
- Pneumatoceles may be a late development, but they rarely require
treatment.
- Most children survive hydrocarbon pneumonitis without complications or
sequelae.
- They are at risk for obstructive pulmonary disease, although results of
long-term pulmonary function studies have been inconclusive.
- Although ingestion of hydrocarbons may cause acute gastrointestinal
irritation: esophagitis and subsequent esophageal stricture are not expected.
- Ingestion of corrosive household
cleaning products is associated with acute
esophagitis and long-term
development of strictures.
Pulmonary hemorrhage has not been
described in hydrocarbon
pneumonitis. Chronic inhalation abuse or "huffing" has been associated
with the sequelae of cardiomyopathy and encephalopathy. An acute effect of
inhalant abuse can be death due to cardiac arrhythmia.
References:
Klein BL, Simon JE. Hydrocarbon poisonings. Pediatr Clin North Am.
1986;33:411-419
Orenstein DM. Aspiration pneumonias and gastroesophageal
reflux-related respiratory disease. In: Behrman RE, Kliegman RM,
Jenson HB, eds. Nelson Textbook of Pediatrics. 16th ed. Philadelphia,
Pa: WB Saunders Co; 2000:1289-1290